ORIGINAL ARTICLE |
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Year : 2019 | Volume
: 3
| Issue : 4 | Page : 236-239 |
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Correlation study between urinary tract bacterial infection and some acute inflammatory responses
Falah Hasan Obayes AL-Khikani1, Ghusin alban Adnan Auda2, Aalae Salman Ayit3
1 Department of Microbiology and Immunology, College of Medicine, Kerbala University, Kerbala, Iraq 2 Department of Microbiology, College of Medical and Health Technology, Baghdad, Iraq 3 Department of Microbiology, Al-Shomally General Hospital, Babil, Iraq
Correspondence Address:
Dr. Falah Hasan Obayes AL-Khikani Department of Microbiology and Immunology, College of Medicine, Kerbala University, Kerbala Iraq
 Source of Support: None, Conflict of Interest: None  | 9 |
DOI: 10.4103/bbrj.bbrj_122_19
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Background: There is no population in the world clear from urinary tract infection (UTI), especially among women. Urinary tract disease is a general term alluding to the bacterial infection anyplace in the urinary tract. It is commonly acknowledged that contamination of the upper urinary tract puts the patient in danger for kidney damage. The aim of the study was to identify the type of bacteria that cause UTI beside elevates the correlation between UTI and some inflammatory markers such as erythrocyte sedimentation rate, white blood cell, C-reactive protein (CRP), and hemoglobin for the UTI among patients in various sex and age groups. Methods: The study was carried in Baghdad Teaching Hospital during July 7, 2017–October 15, 2017. A sum of 45 UTI patients and 20 control group was collected. Results: The study showed that UTI increased in female than in male with 62.2% and 37.8%, respectively, and high risk at age 30–49 years with 42.2%. The microorganisms identified in this study were Escherichia coli (42.2%), followed by Enterobacter (8.9%), Pseudomonas and Klebsiella (6.7% for each), Proteus spp and Serratia spp (4.4% for each), and mixed culture (E. coli + Proteus and E. coli + Pseudomonas with 2.2% for each one). Conclusion: This study showed highly significant correlation between CRP and bacterial isolation.
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